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However, the mechanism by which systemic autoimmunity specifically homes to the joint and bone compartment, thereby triggering inflammation, remains elusive

This could be due to the rapid metabolism and excretion of sulforaphane in vivo (Fahey et al., 2015)

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This creates an integrated feedback mechanism: CHAC1 provides GSH-derived glutamate to support the SLC7A11 antiporter and it promotes NRF2 activation